German Journal "Ärzteblatt" on C-reactive protein
01.04.2022
Today the north-eastern German journal reports on C-reactive protein
01.04.2022
Today the north-eastern German journal reports on C-reactive protein
28.03.2022
A German medical magazine for intensive care and emergency medicine published a review on CRP-Apheresis in patients with acute myocardial infarction and COVID-19
28.03.2022
The Journal of Clinical Medicine published an article on the pros and cons of selective CRP-Apheresis in humans.
26.02.2022
The clinical observations of the article by Fabrizio Esposito, Harald Matthes and Friedemann Schad from the Havelhöhe Community Hospital in Berlin support the hypothesis that CRP is a possible therapeutic target in the treatment of early stages of severe COVID-19.
17.02.2022
A recently published article in the Journal of Clinical Medicine shows the influence of high levels of CRP on G-protein coupled receptors (GPCRs).
30.12.2021
"Most Viewed" article: A new case report from 7 patients suggests the effectiveness of CRP apheresis in severe COVID-19.
02.08.2021
Recently a new case report has been published in Frontiers in Immunology about the first COVID-19 patient at the onset of a severe COVID-19 course. Small heads-up: it turned out amazingly well!
27.07.2021
Another case report has been published on the successful treatment of a young, ventilated COVID-19 patient in Immenstadt. One very effective CRP apheresis was sufficient. Afterwards, the patient's clinical parameters were back to normal.
12.07.2021
In the 3rd issue of "Intensiv-News" 2021, the most recent publications on CRP apheresis were summarized and evaluated with a focus on the recently published CAMI-1 study.
11.03.2021
Results of the first clinical trial of CRP apheresis after acute myocardial infarction (CAMI1) were published in the cardiology journal Frontiers in Cardiovascular Medicine.
"CRP apheresis as anti-inflammatory therapy for acute myocardial infarction: results from the CAMI-1 trial."
The article describes that C-reactive protein substantially triggers myocardial damage after myocardial infarction.